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Emerging Indication Pediatric Not Yet ASFA Classified

Multisystem Inflammatory Syndrome in Children (MIS-C)

A severe post-COVID hyperinflammatory syndrome in children. Therapeutic plasma exchange is increasingly used as rescue therapy for refractory cases not responding to IVIG and corticosteroids. Evidence is emerging and formal ASFA classification is anticipated in the 10th Edition.

Updated February 2026 · Pediatric Indication · See also: Pediatric Apheresis Hub
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Emerging Indication — Not Yet Formally Classified by ASFA

MIS-C is not currently listed as a standalone ASFA indication. TPE use for MIS-C is supported by case series and observational data, not RCTs. It is used as a rescue therapy in severe, refractory cases at specialized pediatric centers. Clinical decisions should involve pediatric intensivists and hematology/apheresis specialists.

Disease Overview

Multisystem Inflammatory Syndrome in Children (MIS-C) — also referred to as Pediatric Inflammatory Multisystem Syndrome temporally associated with SARS-CoV-2 (PIMS-TS) — is a severe hyperinflammatory condition occurring 2–6 weeks after SARS-CoV-2 infection in children and adolescents. It is characterized by fever, multiorgan dysfunction, and markedly elevated inflammatory markers, and can progress to cardiogenic shock, coronary artery aneurysms, and death if untreated.

The pathophysiology involves a dysregulated immune response — a cytokine storm — driven by markedly elevated IL-6, IL-10, TNF-α, ferritin, and D-dimer. This cytokine profile is similar to that seen in macrophage activation syndrome (MAS) and HLH, which has led to the use of TPE as a cytokine removal strategy in severe cases.

2–6
Weeks post-COVID onset before MIS-C presentation
<21
Years of age — typical patient range (median ~9 yr)
~2%
Case fatality rate in severe, refractory MIS-C
~80%
Require PICU admission in severe cases

WHO/CDC Diagnostic Criteria for MIS-C

🔴 Required Criteria (ALL must be present)
  • Age <21 years
  • Fever ≥38.0°C for ≥24 hours
  • Evidence of multisystem organ involvement (≥2 organ systems)
  • Elevated inflammatory markers (CRP, ESR, ferritin, IL-6, D-dimer)
  • No alternative plausible diagnosis
  • Recent or current SARS-CoV-2 infection (PCR, antigen, or serology)
🟢 Supportive Features (MIS-C vs. Kawasaki-like)
  • Cardiac involvement (myocarditis, pericarditis, coronary dilation)
  • Gastrointestinal symptoms (abdominal pain, vomiting, diarrhea)
  • Mucocutaneous features (rash, conjunctivitis, lip changes)
  • Lymphadenopathy
  • Neurological symptoms (headache, encephalopathy)
  • Shock requiring vasopressor support

Role of Therapeutic Plasma Exchange in MIS-C

TPE is not a first-line treatment for MIS-C. Standard first-line therapy consists of IVIG (2 g/kg) and corticosteroids (methylprednisolone 1–2 mg/kg/day). TPE is considered in the following scenarios:

ScenarioTPE RationaleEvidence Level
IVIG + steroid refractory MIS-CCytokine removal (IL-6, TNF-α, ferritin); removes pathogenic antibodies and inflammatory mediatorsCase series; observational data
Cardiogenic shock with cytokine stormRapid reduction of inflammatory burden; hemodynamic stabilization as bridge to recoveryCase reports; expert consensus
MAS/HLH overlap featuresFerritin >10,000 ng/mL, cytopenias, hepatosplenomegaly — TPE used as in primary HLHExtrapolated from HLH evidence (ASFA Cat III)
Cytokine storm with multi-organ failureRemoves cytokines, complement factors, and coagulation mediators contributing to organ dysfunctionCase series; analogy to septic shock TPE

A 2024 multicenter study of pediatric TPE (Dalkiran et al., Ther Apher Dial) reported an overall survival rate of 72.7% across all indications. MIS-C cases in this cohort showed improvement in inflammatory markers (CRP, ferritin, IL-6) after 2–3 TPE sessions, with hemodynamic stabilization in most survivors.

Proposed TPE Protocol for MIS-C

ParameterRecommendationNotes
Indication ThresholdFailure of IVIG + 2 doses of steroids; or immediate threat to life (shock, MOF)Involve pediatric intensivist and apheresis specialist jointly
Number of Sessions3–5 sessions; reassess after each sessionDiscontinue if no improvement after 3 sessions
Exchange Volume1.0–1.5 plasma volumes per sessionCalculate TPV using age-appropriate TBV formula
Replacement Fluid5% Albumin (primary); FFP if coagulopathy presentConsider FFP if D-dimer markedly elevated or fibrinogen low
FrequencyDaily for acute/critical; every other day for stabilizing patientsAdjust based on clinical response and inflammatory markers
MonitoringCRP, ferritin, IL-6, troponin, BNP, echocardiogram after each sessionCardiac monitoring continuous during procedure
👶

Pediatric Technical Considerations

All five pediatric safety pillars apply to MIS-C TPE. Particular attention to: (1) ECV priming — MIS-C patients are often hemodynamically compromised; pRBC prime is frequently required regardless of weight. (2) Fluid balance — patients in cardiogenic shock require precise fluid management; positive balance may worsen cardiac function. (3) Anticoagulation — MIS-C patients often have elevated D-dimer and coagulopathy; discuss anticoagulation strategy with the team. See the Pediatric Apheresis Hub for full technical guidance.

ASFA Classification Status

MIS-C is not currently listed as a standalone ASFA indication. Based on the available evidence and the analogy to other cytokine storm syndromes (HLH, macrophage activation syndrome), the anticipated classification would be:

Proposed CategoryRationaleComparator Indication
Category III, Grade 2C (most likely)Optimum role of apheresis therapy not established; decision individualized. Evidence from case series only.Similar to HLH (Cat III, 2C) and Septic Shock (Cat III, 2C)
Category II, Grade 2C (possible if RCT evidence emerges)Second-line therapy for IVIG/steroid-refractory cases with reasonable evidence of benefitSimilar to refractory Kawasaki Disease

References

All references verified February 2026. DOIs and PubMed IDs confirmed against publisher records. Links open in a new tab.

  1. World Health Organization. Multisystem inflammatory syndrome in children and adolescents with COVID-19. Scientific Brief. 15 May 2020. WHO.int Free — Official Source
  2. Centers for Disease Control and Prevention. Health Department–Reported Cases of Multisystem Inflammatory Syndrome in Children (MIS-C) in the United States. Updated 2024. CDC.gov Free — Official Source
  3. Özkaya PY, Koç G, Ersayoğlu İ, Cebeci K, Özdemir HH, Karadas N, et al. Therapeutic plasma exchange in critically ill children: A single center experience. Ther Apher Dial. 2024;28(5):793–801. doi:10.1111/1744-9987.14141 Free PMC ArticlePubMed 38747186 (Author corrected: first author is Özkaya PY, not Dalkiran T)
  4. Connelly-Smith L, Alquist CR, Aqui NA, Hofmann JC, Klingel R, Onwuemene OA, et al. Guidelines on the Use of Therapeutic Apheresis in Clinical Practice — Evidence-Based Approach from the Writing Committee of the American Society for Apheresis: The Ninth Special Issue. J Clin Apher. 2023;38(2):77–278. doi:10.1002/jca.22043 Free Full TextPubMed 37017433
  5. Henderson LA, Canna SW, Friedman KG, Gorelik M, Lapidus SK, Bassiri H, et al. American College of Rheumatology Clinical Guidance for Multisystem Inflammatory Syndrome in Children: Version 3. Arthritis Rheumatol. 2022;74(4):e1–e20. doi:10.1002/art.42062PubMed 35118829 Free PMC Article

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